BNIP3 (Bcl-2 19 kDa Interacting Protein) Acts as Transcriptional Repressor of Apoptosis-Inducing Factor Expression Preventing Cell Death in Human Malignant Gliomas

Ítems similars

• BNIP3 acts as transcriptional repressor of death receptor-5 expression and prevents TRAIL-induced cell death in gliomas
  per: Burton, T R, et al.
  Publicat: (2013)
• The role of Bcl-2 family member BNIP3 in cell death and disease: NIPping at the heels of cell death
  per: Burton, Teralee R., et al.
  Publicat: (2009)
• The pro-cell death Bcl-2 family member, BNIP3, is localized to the nucleus of human glial cells: Implications for glioblastoma multiforme tumor cell survival under hypoxia
  per: Burton, Teralee R., et al.
  Publicat: (2006)
• Truncated forms of BNIP3 act as dominant negatives inhibiting hypoxia-induced cell death
  per: Bristow, Nicolle, et al.
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• Bcl-2 family member Mcl-1 expression is reduced under hypoxia by the E3 ligase FBW7 contributing to BNIP3 induced cell death in glioma cells
  per: Chen, Yongqiang, et al.
  Publicat: (2015)