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Transgenic inhibition of Nogo-66 receptor function allows axonal sprouting and improved locomotion after spinal injury
Axon growth after spinal injury is thought to be limited in part by myelin-derived proteins that act via the Nogo-66 Receptor (NgR). To test this hypothesis, we sought to study recovery from spinal cord injury (SCI) after inhibiting NgR transgenically with a soluble function-blocking NgR fragment. G...
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| Hoofdauteurs: | , , , , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
2005
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3246391/ https://ncbi.nlm.nih.gov/pubmed/15866044 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.mcn.2004.12.008 |
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