Delayed Systemic Nogo-66 Receptor Antagonist Promotes Recovery from Spinal Cord Injury

Traumatized axons possess an extremely limited ability to regenerate within the adult mammalian CNS. The myelin-derived axon outgrowth inhibitors Nogo, oligodendrocyte-myelin glycoprotein, and myelin-associated glycoprotein, all bind to an axonal Nogo-66 receptor (NgR) and at least partially account...

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Bibliografiset tiedot
Julkaisussa:J Neurosci
Päätekijät: Li, Shuxin, Strittmatter, Stephen M.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Society for Neuroscience 2003
Aiheet:
Development/Plasticity/Repair
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC6741116/
https://ncbi.nlm.nih.gov/pubmed/12764110
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.23-10-04219.2003
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