Whole-exome sequencing uncovers frequent GNAS mutations in intraductal papillary mucinous neoplasms of the pancreas

Míreanna Comhchosúla

• Targeting of MAPK-associated molecules identifies SON as a prime target to attenuate the proliferation and tumorigenicity of pancreatic cancer cells
  le: Furukawa, Toru, et al.
  Foilsithe: (2012)
• A GNAS Mutation Found in Pancreatic Intraductal Papillary Mucinous Neoplasms Induces Drastic Alterations of Gene Expression Profiles with Upregulation of Mucin Genes
  le: Komatsu, Hirotake, et al.
  Foilsithe: (2014)
• Whole exome sequencing reveals recurrent mutations in BRCA2 and FAT genes in acinar cell carcinomas of the pancreas
  le: Furukawa, Toru, et al.
  Foilsithe: (2015)
• Frequent GNAS mutations in low-grade appendiceal mucinous neoplasms
  le: Nishikawa, G, et al.
  Foilsithe: (2013)
• Clinicopathological Correlates of Activating GNAS Mutations in Intraductal Papillary Mucinous Neoplasm (IPMN) of the Pancreas
  le: Molin, Marco Dal, et al.
  Foilsithe: (2013)