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C-terminal tetrapeptides inhibit Aβ42-induced neurotoxicity primarily through specific interaction at the N-terminus of Aβ42
Inhibition of amyloid β-protein (Aβ)-induced toxicity is a promising therapeutic strategy for Alzheimer’s disease (AD). Previously, we reported that the C-terminal tetrapeptide Aβ(39–42) is a potent inhibitor of neurotoxicity caused by Aβ42, the form of Aβ most closely associated with AD. Here, init...
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| Main Authors: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2011
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3240737/ https://ncbi.nlm.nih.gov/pubmed/22087474 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1021/jm200982p |
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