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Transmembrane mutations in Toll-like Receptor 9 bypass the requirement for ectodomain proteolysis and induce fatal inflammation

Recognition of nucleic acids as a signature of infection by Toll-like receptors (TLRs) 7 and 9 exposes the host to potential self-recognition and autoimmunity. It has been proposed that intracellular compartmentalization is largely responsible for reliable self versus non-self discrimination by thes...

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Detalhes bibliográficos
Main Authors: Mouchess, Maria L., Arpaia, Nicholas, Souza, Gianne, Barbalat, Roman, Ewald, Sarah E., Lau, Laura, Barton, Gregory M.
Formato: Artigo
Idioma:Inglês
Publicado em: 2011
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3230302/
https://ncbi.nlm.nih.gov/pubmed/22078797
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.immuni.2011.10.009
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