Deficient degradation of homotrimeric type I collagen,α1(I)(3) glomerulopathy in oim mice

Col1a2-deficient (oim) mice synthesize homotrimeric type I collagen due to nonfunctional proα2(I) collagen chains. Our previous studies revealed a postnatal, progressive type I collagen glomerulopathy in this mouse model, but the mechanism of the sclerotic collagen accumulation within the renal mesa...

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Autors principals: Roberts-Pilgrim, Anna M., Makareeva, Elena, Myles, Matthew H., Besch-Williford, Cynthia L., Brodeur, Amanda C., Walker, Andrew L., Leikin, Sergey, Franklin, Craig L., Phillips, Charlotte L.
Format: Artigo
Idioma:Inglês
Publicat: 2011
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Article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3205245/
https://ncbi.nlm.nih.gov/pubmed/21855382
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ymgme.2011.07.025
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