Imatinib sensitivity in BCR-ABL1-positive chronic myeloid leukemia cells is regulated by the remaining normal ABL1 allele

Lignende værker

• Monoubiquitinated Fanconi Anemia D2 (FANCD2-Ub) Is Required for BCR-ABL1 Kinase -Induced Leukemogenesis
  af: Koptyra, Mateusz, et al.
  Udgivet: (2011)
• BCR/ABL kinase induces self-mutagenesis via reactive oxygen species to encode imatinib resistance
  af: Koptyra, Mateusz, et al.
  Udgivet: (2006)
• The PERK-eIF2α phosphorylation arm is a pro-survival pathway of BCR-ABL signaling and confers resistance to imatinib treatment in chronic myeloid leukemia cells
  af: Kusio-Kobialka, Monika, et al.
  Udgivet: (2012)
• BCR/ABL modifies the kinetics and fidelity of DNA double-strand breaks repair in hematopoietic cells
  af: Slupianek, Artur, et al.
  Udgivet: (2005)
• Reactive Oxygen Species and Mitochondrial DNA Damage and Repair in BCR-ABL1 Cells Resistant to Imatinib
  af: Blasiak, Janusz, et al.
  Udgivet: (2015)