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TAT-μUtrophin mitigates the pathophysiology of dystrophin and utrophin double-knockout mice
Previously, we demonstrated functional substitution of dystrophin by TAT-μUtrophin (TAT-μUtr) in dystrophin-deficient mdx mice. Herein, we addressed whether TAT-μUtr could improve the phenotype of dystrophin and utrophin double-knockout (mdx:utr(−/−)) mice. Specifically, we quantitatively compared s...
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| Hlavní autoři: | , , |
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| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
American Physiological Society
2011
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3137527/ https://ncbi.nlm.nih.gov/pubmed/21565990 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/japplphysiol.00248.2011 |
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