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TAT-μUtrophin mitigates the pathophysiology of dystrophin and utrophin double-knockout mice

Previously, we demonstrated functional substitution of dystrophin by TAT-μUtrophin (TAT-μUtr) in dystrophin-deficient mdx mice. Herein, we addressed whether TAT-μUtr could improve the phenotype of dystrophin and utrophin double-knockout (mdx:utr(−/−)) mice. Specifically, we quantitatively compared s...

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Wedi'i Gadw mewn:
Manylion Llyfryddiaeth
Prif Awduron: Call, Jarrod A., Ervasti, James M., Lowe, Dawn A.
Fformat: Artigo
Iaith:Inglês
Cyhoeddwyd: American Physiological Society 2011
Pynciau:
Mynediad Ar-lein:https://ncbi.nlm.nih.gov/pmc/articles/PMC3137527/
https://ncbi.nlm.nih.gov/pubmed/21565990
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/japplphysiol.00248.2011
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