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Requirement of c-Jun NH(2)-Terminal Kinase for Ras-Initiated Tumor Formation
The c-Jun NH(2)-terminal kinase (JNK) signal transduction pathway causes increased gene expression mediated, in part, by members of the activating transcription factor protein (AP1) group. JNK is therefore implicated in the regulation of cell growth and cancer. To test the role of JNK in Ras-induced...
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| 主要な著者: | , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Microbiology
2011
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3135291/ https://ncbi.nlm.nih.gov/pubmed/21282468 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.01122-10 |
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