Amyloid β-Induced Impairments in Hippocampal Synaptic Plasticity Are Rescued by Decreasing Mitochondrial Superoxide

Generation of reactive oxygen species (ROS) causes cellular oxidative damage and has been implicated in the etiology of Alzheimer's disease (AD). In contrast, multiple lines of evidence indicate that ROS can normally modulate long-term potentiation (LTP), a cellular model for memory formation....

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Autors principals: Ma, Tao, Hoeffer, Charles A., Wong, Helen, Massaad, Cynthia A., Zhou, Ping, Iadecola, Costantino, Murphy, Michael P., Pautler, Robia G., Klann, Eric
Format: Artigo
Idioma:Inglês
Publicat: Society for Neuroscience 2011
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Articles
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3095121/
https://ncbi.nlm.nih.gov/pubmed/21490199
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.6566-10.2011
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