Amyloid β-Induced Impairments in Hippocampal Synaptic Plasticity Are Rescued by Decreasing Mitochondrial Superoxide

Generation of reactive oxygen species (ROS) causes cellular oxidative damage and has been implicated in the etiology of Alzheimer's disease (AD). In contrast, multiple lines of evidence indicate that ROS can normally modulate long-term potentiation (LTP), a cellular model for memory formation....

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Main Authors: Ma, Tao, Hoeffer, Charles A., Wong, Helen, Massaad, Cynthia A., Zhou, Ping, Iadecola, Costantino, Murphy, Michael P., Pautler, Robia G., Klann, Eric
格式: Artigo
語言:Inglês
出版: Society for Neuroscience 2011
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在線閱讀:https://ncbi.nlm.nih.gov/pmc/articles/PMC3095121/
https://ncbi.nlm.nih.gov/pubmed/21490199
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.6566-10.2011
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