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Soluble guanylate cyclase-α(1) is required for the cardioprotective effects of inhaled nitric oxide
Reperfusion injury limits the benefits of revascularization in the treatment of myocardial infarction (MI). Breathing nitric oxide (NO) reduces cardiac ischemia-reperfusion injury in animal models; however, the signaling pathways by which inhaled NO confers cardioprotection remain uncertain. The obj...
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| 主要な著者: | , , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Physiological Society
2011
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3075043/ https://ncbi.nlm.nih.gov/pubmed/21257915 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00948.2010 |
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