Soluble guanylate cyclase-α(1) is required for the cardioprotective effects of inhaled nitric oxide

Reperfusion injury limits the benefits of revascularization in the treatment of myocardial infarction (MI). Breathing nitric oxide (NO) reduces cardiac ischemia-reperfusion injury in animal models; however, the signaling pathways by which inhaled NO confers cardioprotection remain uncertain. The obj...

ver descrição completa

Na minha lista:
Detalhes bibliográficos
Main Authors: Nagasaka, Yasuko, Buys, Emmanuel S., Spagnolli, Ester, Steinbicker, Andrea U., Hayton, Sarah R., Rauwerdink, Kristen M., Brouckaert, Peter, Zapol, Warren M., Bloch, Kenneth D.
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2011
Assuntos:
Integrative Cardiovascular Physiology and Pathophysiology
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3075043/
https://ncbi.nlm.nih.gov/pubmed/21257915
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00948.2010
Tags: Adicionar Tag
Sem tags, seja o primeiro a adicionar uma tag!

Registos relacionados