Memory Deficits Due to Familial British Dementia BRI2 Mutation Are Caused by Loss of BRI2 Function Rather than Amyloidosis

Lignende værker

• APP heterozygosity averts memory deficit in knockin mice expressing the Danish dementia BRI2 mutant
  af: Tamayev, Robert, et al.
  Udgivet: (2011)
• β- but not γ-secretase proteolysis of APP causes synaptic and memory deficits in a mouse model of dementia
  af: Tamayev, Robert, et al.
  Udgivet: (2012)
• Memory Deficits of British Dementia Knock-In Mice Are Prevented by Aβ-Precursor Protein Haploinsufficiency
  af: Tamayev, Robert, et al.
  Udgivet: (2012)
• Danish dementia mice suggest that loss of function and not the amyloid cascade causes synaptic plasticity and memory deficits
  af: Tamayev, Robert, et al.
  Udgivet: (2010)
• Danish and British dementia ITM2b/BRI2 mutations reduce BRI2 protein stability and impair glutamatergic synaptic transmission
  af: Yin, Tao, et al.
  Udgivet: (2020)