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Mechanism of rescue of common p53 cancer mutations by second-site suppressor mutations

The core domain of p53 is extremely susceptible to mutations that lead to loss of function. We analysed the stability and DNA-binding activity of such mutants to understand the mechanism of second-site suppressor mutations. Double-mutant cycles show that N239Y and N268D act as ‘global stability’ sup...

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Bibliografiske detaljer
Main Authors: Nikolova, Penka V., Wong, Kam-Bo, DeDecker, Brian, Henckel, Julia, Fersht, Alan R.
Format: Artigo
Sprog:Inglês
Udgivet: Oxford University Press 2000
Fag:
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC305574/
https://ncbi.nlm.nih.gov/pubmed/10654936
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/emboj/19.3.370
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