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Amyloid-β42 Alters Apolipoprotein E Solubility in Brains of Mice with Five Familial AD Mutations
Amyloid plaques composed of the 42 amino acid form of amyloid-beta peptide (Aβ42) are a pathological hallmark of Alzheimer’s disease (AD), but soluble and intraneuronal Aβ42 are the more proximal causes of synaptic dysfunction and neurotoxicity. Apolipoprotein E (apoE) modulates this disease process...
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| 主要な著者: | , , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2011
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3049315/ https://ncbi.nlm.nih.gov/pubmed/21219931 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jneumeth.2010.12.025 |
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