β-Cell Dysfunctional ERAD/Ubiquitin/Proteasome System in Type 2 Diabetes Mediated by Islet Amyloid Polypeptide–Induced UCH-L1 Deficiency

Ítems similars

• UCHL1 deficiency exacerbates human islet amyloid polypeptide toxicity in β-cells: Evidence of interplay between the ubiquitin/proteasome system and autophagy
  per: Costes, Safia, et al.
  Publicat: (2014)
• The effect of curcumin on human islet amyloid polypeptide misfolding and toxicity
  per: DAVAL, MARIE, et al.
  Publicat: (2010)
• Autophagy defends pancreatic β cells from human islet amyloid polypeptide-induced toxicity
  per: Rivera, Jacqueline F., et al.
  Publicat: (2014)
• β Cell–specific increased expression of calpastatin prevents diabetes induced by islet amyloid polypeptide toxicity
  per: Gurlo, Tatyana, et al.
  Publicat: (2016)
• Beneficial Endocrine but Adverse Exocrine Effects of Sitagliptin in the Human Islet Amyloid Polypeptide Transgenic Rat Model of Type 2 Diabetes: Interactions With Metformin
  per: Matveyenko, Aleksey V., et al.
  Publicat: (2009)