Ablation of Skeletal Muscle Triadin Impairs FKBP12/RyR1 Channel Interactions Essential for Maintaining Resting Cytoplasmic Ca(2+)

Previously, we have shown that lack of expression of triadins in skeletal muscle cells results in significant increase of myoplasmic resting free Ca(2+) ([Ca(2+)](rest)), suggesting a role for triadins in modulating global intracellular Ca(2+) homeostasis. To understand this mechanism, we study here...

Descripción completa

Guardado en:
Detalles Bibliográficos
Autores principales: Eltit, Jose M., Feng, Wei, Lopez, Jose R., Padilla, Isela T., Pessah, Isaac N., Molinski, Tadeusz F., Fruen, Bradley R., Allen, Paul D., Perez, Claudio F.
Formato: Artigo
Lenguaje:Inglês
Publicado: American Society for Biochemistry and Molecular Biology 2010
Materias:
Cell Biology
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC2992278/
https://ncbi.nlm.nih.gov/pubmed/20926377
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M110.164525
Etiquetas: Agregar Etiqueta
Sin Etiquetas, Sea el primero en etiquetar este registro!

Ejemplares similares