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Reduced gain of excitation contraction coupling in triadin-null myotubes is mediated by the disruption of FKBP12/RyR1 interaction
Several studies have suggested that triadin (Tdn) may be a critical component of skeletal EC-coupling. However, using Tdn-null mice we have shown that triadin ablation results in no significant disruption of skeletal EC-coupling. To analyze the role of triadin in EC-coupling signaling here we used w...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2011
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3052628/ https://ncbi.nlm.nih.gov/pubmed/21310482 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ceca.2011.01.005 |
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