Ablation of Skeletal Muscle Triadin Impairs FKBP12/RyR1 Channel Interactions Essential for Maintaining Resting Cytoplasmic Ca(2+)

Previously, we have shown that lack of expression of triadins in skeletal muscle cells results in significant increase of myoplasmic resting free Ca(2+) ([Ca(2+)](rest)), suggesting a role for triadins in modulating global intracellular Ca(2+) homeostasis. To understand this mechanism, we study here...

Description complète

Enregistré dans:
Détails bibliographiques
Auteurs principaux: Eltit, Jose M., Feng, Wei, Lopez, Jose R., Padilla, Isela T., Pessah, Isaac N., Molinski, Tadeusz F., Fruen, Bradley R., Allen, Paul D., Perez, Claudio F.
Format: Artigo
Langue:Inglês
Publié: American Society for Biochemistry and Molecular Biology 2010
Sujets:
Cell Biology
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC2992278/
https://ncbi.nlm.nih.gov/pubmed/20926377
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M110.164525
Tags: Ajouter un tag
Pas de tags, Soyez le premier à ajouter un tag!

Documents similaires