Ablation of Skeletal Muscle Triadin Impairs FKBP12/RyR1 Channel Interactions Essential for Maintaining Resting Cytoplasmic Ca(2+)

Previously, we have shown that lack of expression of triadins in skeletal muscle cells results in significant increase of myoplasmic resting free Ca(2+) ([Ca(2+)](rest)), suggesting a role for triadins in modulating global intracellular Ca(2+) homeostasis. To understand this mechanism, we study here...

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Detalhes bibliográficos
Main Authors: Eltit, Jose M., Feng, Wei, Lopez, Jose R., Padilla, Isela T., Pessah, Isaac N., Molinski, Tadeusz F., Fruen, Bradley R., Allen, Paul D., Perez, Claudio F.
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Biochemistry and Molecular Biology 2010
Assuntos:
Cell Biology
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2992278/
https://ncbi.nlm.nih.gov/pubmed/20926377
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M110.164525
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