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Hemoglobin synthesis in β-thalassemia: the properties of the free α-chains

The decrease in hemoglobin A (HbA, α(2)β(2)) synthesis in the erythroid cells of patients with β-thalassemia is due to a selective defect in β-chain synthesis. Since α-chains continue to be formed at a normal rate in these cells, this results in a marked relative excess of α-chain synthesis over β-...

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Bibliografiske detaljer
Hovedforfatter: Bank, Arthur
Format: Artigo
Sprog:Inglês
Udgivet: 1968
Fag:
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC297235/
https://ncbi.nlm.nih.gov/pubmed/5641622
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