Hemoglobin synthesis in β-thalassemia: the properties of the free α-chains
The decrease in hemoglobin A (HbA, α(2)β(2)) synthesis in the erythroid cells of patients with β-thalassemia is due to a selective defect in β-chain synthesis. Since α-chains continue to be formed at a normal rate in these cells, this results in a marked relative excess of α-chain synthesis over β-...
שמור ב:
| הוצא לאור ב: | J Clin Invest |
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| מחבר ראשי: | |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
American Society for Clinical Investigation
1968
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC297235/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/5641622/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI105779 |
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