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Oligodendrocyte PTEN required for myelin and axonal integrity not remyelination

OBJECTIVE: Repair of myelin injury in multiple sclerosis may fail resulting in chronic demyelination, axonal loss and disease progression. As cellular pathways regulated by Phosphatase and tensin homologue deleted on chromosome 10 (PTEN; e.g., PI-3Kinase) have been reported to enhance axon regenerat...

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書誌詳細
主要な著者: Harrington, Emily P., Zhao, Chao, Fancy, Stephen P.J., Kaing, Sovann, Franklin, Robin J.M., Rowitch, David H.
フォーマット: Artigo
言語:Inglês
出版事項: 2010
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC2966537/
https://ncbi.nlm.nih.gov/pubmed/20853437
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/ana.22090
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