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NOD2 and TLR2 function independently in a murine model of arthritis triggered by intra-articular peptidoglycan
OBJECTIVE: Blau syndrome is an autoinflammatory disease resulting from mutations in NOD2 (nucleotide-binding oligomerization domain 2), wherein granulomatous arthritis, uveitis and dermatitis develop. The mechanisms by which aberrant NOD2 causes joint inflammation are poorly understood. Indeed very...
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| Main Authors: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2010
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2945367/ https://ncbi.nlm.nih.gov/pubmed/20131263 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/art.27335 |
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