Insulin secretory abnormalities in subjects with hyperglycemia due to glucokinase mutations.

Pancreatic beta-cell function was studied in six subjects with mutations in the enzyme glucokinase (GCK) who were found to have elevated fasting and postprandial glucose levels in comparison to six normoglycemic controls. Insulin secretion rates (ISRs) were estimated by deconvolution of peripheral C...

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Bibliografische gegevens
Gepubliceerd in:	J Clin Invest
Hoofdauteurs:	Byrne, M M, Sturis, J, Clément, K, Vionnet, N, Pueyo, M E, Stoffel, M, Takeda, J, Passa, P, Cohen, D, Bell, G I
Formaat:	Artigo
Taal:	Inglês
Gepubliceerd in:	American Society for Clinical Investigation 1994
Onderwerpen:	Research Article
Online toegang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC294056/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8132752/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI117064
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Insulin secretory abnormalities in subjects with hyperglycemia due to glucokinase mutations.

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