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XPA impacts formation but not proteasome-sensitive repair of DNA–protein cross-links induced by chromate
DNA–protein cross-links (DPCs) are caused by a large number of human carcinogens and anti-cancer drugs. However, cellular processes involved in decreasing a burden of these genotoxic lesions remain poorly understood. Here, we examined the impact of nucleotide excision repair (NER), which is a princi...
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| Main Authors: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Oxford University Press
2010
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2893307/ https://ncbi.nlm.nih.gov/pubmed/20410141 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/mutage/geq017 |
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