XPA impacts formation but not proteasome-sensitive repair of DNA–protein cross-links induced by chromate

DNA–protein cross-links (DPCs) are caused by a large number of human carcinogens and anti-cancer drugs. However, cellular processes involved in decreasing a burden of these genotoxic lesions remain poorly understood. Here, we examined the impact of nucleotide excision repair (NER), which is a princi...

Täydet tiedot

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Bibliografiset tiedot
Päätekijät: Zecevic, Alma, Hagan, Elizabeth, Reynolds, Mindy, Poage, Graham, Johnston, Tatiana, Zhitkovich, Anatoly
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Oxford University Press 2010
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Original Articles
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC2893307/
https://ncbi.nlm.nih.gov/pubmed/20410141
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/mutage/geq017
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