Aβ oligomers inhibit synapse remodelling necessary for memory consolidation

Extensive research has implicated the amyloid-β protein (Aβ) in the aetiology of Alzheimer’s disease (AD). This protein has been shown to produce memory deficits when injected into rodent brain and in mouse models of AD Aβ production is associated with impaired learning and/or recall. Here we examin...

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Autores principales: Freir, Darragh B., Fedriani, Rocio, Scully, Darren, Smith, Imelda M., Selkoe, Dennis J., Walsh, Dominic M., Regan, Ciaran M.
Formato: Artigo
Lenguaje:Inglês
Publicado: 2010
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Article
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC2891223/
https://ncbi.nlm.nih.gov/pubmed/20097446
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neurobiolaging.2010.01.001
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