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Complement drives Th17 cell differentiation and triggers autoimmune arthritis
Activation of serum complement triggers Th17 cell–dependent spontaneous autoimmune disease in an animal model. In genetically autoimmune-prone SKG mice, administration of mannan or β-glucan, both of which activate serum complement, evoked Th17 cell–mediated chronic autoimmune arthritis. C5a, a chief...
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| Main Authors: | , , , , , , , , , , , , , , |
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| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
The Rockefeller University Press
2010
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2882841/ https://ncbi.nlm.nih.gov/pubmed/20457757 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1084/jem.20092301 |
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