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Deletion of puma protects hippocampal neurons in a model of severe status epilepticus

Prolonged seizures (status epilepticus) can activate apoptosis-associated signaling pathways. The extent to which such pathways contribute to cell death might depend on the insult intensity, whereby the programmed or apoptotic cell death component is reduced when seizures are more severe or protract...

詳細記述

保存先:
書誌詳細
主要な著者: Engel, Tobias, Hatazaki, Seiji, Tanaka, Katsuhiro, Prehn, Jochen H.M., Henshall, David C.
フォーマット: Artigo
言語:Inglês
出版事項: 2010
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC2877385/
https://ncbi.nlm.nih.gov/pubmed/20362645
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuroscience.2010.03.057
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