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Deletion of puma protects hippocampal neurons in a model of severe status epilepticus

Prolonged seizures (status epilepticus) can activate apoptosis-associated signaling pathways. The extent to which such pathways contribute to cell death might depend on the insult intensity, whereby the programmed or apoptotic cell death component is reduced when seizures are more severe or protract...

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Detalhes bibliográficos
Main Authors: Engel, Tobias, Hatazaki, Seiji, Tanaka, Katsuhiro, Prehn, Jochen H.M., Henshall, David C.
Formato: Artigo
Idioma:Inglês
Publicado em: 2010
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2877385/
https://ncbi.nlm.nih.gov/pubmed/20362645
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuroscience.2010.03.057
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