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A single mutation promotes amyloidogenicity through a highly promiscuous dimer interface
Light chain amyloidosis is a devastating protein misfolding disease characterized by the accumulation of amyloid fibrils that causes tissue damage and organ failure. These fibrils are composed of monoclonal light chain protein secreted from an abnormal proliferation of bone marrow plasma cells. We p...
Gorde:
| Egile Nagusiak: | , , , , |
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| Formatua: | Artigo |
| Hizkuntza: | Inglês |
| Argitaratua: |
2010
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| Gaiak: | |
| Sarrera elektronikoa: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2872106/ https://ncbi.nlm.nih.gov/pubmed/20462490 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.str.2010.02.012 |
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