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Mutant huntingtin fragment selectively suppresses Brn-2 POU domain transcription factor to mediate hypothalamic cell dysfunction

In polyglutamine diseases including Huntington's disease (HD), mutant proteins containing expanded polyglutamine stretches form nuclear aggregates in neurons. Although analysis of their disease models suggested a significance of transcriptional dysregulation in these diseases, how it mediates t...

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Autors principals: Yamanaka, Tomoyuki, Tosaki, Asako, Miyazaki, Haruko, Kurosawa, Masaru, Furukawa, Yoshiaki, Yamada, Mizuki, Nukina, Nobuyuki
Format: Artigo
Idioma:Inglês
Publicat: Oxford University Press 2010
Matèries:
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC2865370/
https://ncbi.nlm.nih.gov/pubmed/20185558
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/hmg/ddq087
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