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Interleukin-17 regulates CXCL1 mRNA stability via an AUUUA/Tristetraprolin-independent sequence
Interleukin (IL)-17 contributes to inflammatory response in part by promoting enhanced expression of chemokines such as CXCL1 by prolonging the half-life of this constitutively unstable mRNA. While IL-17 is a weak stimulus for transcription of the CXCL1 gene, it strongly potentiates message accumula...
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| Autors principals: | , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2009
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2829999/ https://ncbi.nlm.nih.gov/pubmed/20042592 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.0902423 |
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