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Bcl2 Negatively Regulates DNA Double-Strand Break Repair through a Non-Homologous End Joining Pathway

Bcl2 can enhance susceptibility to carcinogenesis but the mechanism(s) remains fragmentary. Here we discovered that Bcl2 suppresses DNA double-strand break (DSB) repair and V(D)J recombination by down-regulating Ku DNA binding activity, which is associated with increased genetic instability. Exposur...

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Autori principali: Wang, Qinhong, Gao, Fengqin, May, W. Stratford, Zhang, Yangde, Flagg, Tammy, Deng, Xingming
Natura: Artigo
Lingua:Inglês
Pubblicazione: 2008
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC2806186/
https://ncbi.nlm.nih.gov/pubmed/18313386
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.molcel.2007.12.029
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