Bone morphogenetic protein signaling is impaired in an Hfe knockout mouse model of hemochromatosis

BACKGROUND AND AIMS: Mutations in HFE are the most common cause of the iron-overload disorder hereditary hemochromatosis (HH). Levels of the main iron regulatory hormone, hepcidin, are inappropriately low in HH mouse models and patients with HFE mutations, indicating that HFE regulates hepcidin. The...

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Autors principals: Corradini, Elena, Garuti, Cinzia, Montosi, Giuliana, Ventura, Paolo, Andriopoulos, Billy, Lin, Herbert Y., Pietrangelo, Antonello, Babitt, Jodie L.
Format: Artigo
Idioma:Inglês
Publicat: 2009
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Article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC2757523/
https://ncbi.nlm.nih.gov/pubmed/19591830
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1053/j.gastro.2009.06.057
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