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Renal and Brain Isoforms of WNK3 Have Opposite Effects on NCCT Expression

Mutations in the WNK kinases WNK1 and WNK4 cause a rare familial form of hypertension (Gordon syndrome) by increasing expression of the thiazide-sensitive co-transporter NCCT in the kidney. Regulation of NCCT expression involves a scaffold of proteins composed of several kinases, including the third...

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Autores principales: Glover, Mark, Zuber, Annie Mercier, O’Shaughnessy, Kevin M.
Formato: Artigo
Lenguaje:Inglês
Publicado: American Society of Nephrology 2009
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Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC2689907/
https://ncbi.nlm.nih.gov/pubmed/19470686
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2008050542
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