Mice that lack matrix metalloproteinase-9 display delayed wound healing associated with delayed reepithelization and disordered collagen fibrillogenesis

Samankaltaisia teoksia

• Macrophage fusion, giant cell formation, and the foreign body response require matrix metalloproteinase 9
  Tekijä: MacLauchlan, Susan, et al.
  Julkaistu: (2009)
• Mice That Lack Thrombospondin 2 Display Connective Tissue Abnormalities That Are Associated with Disordered Collagen Fibrillogenesis, an Increased Vascular Density, and a Bleeding Diathesis
  Tekijä: Kyriakides, Themis R., et al.
  Julkaistu: (1998)
• Lack of TNF-α–Induced MMP-9 Production and Abnormal E-Cadherin Redistribution Associated with Compromised Fusion in MCP-1–Null Macrophages
  Tekijä: Skokos, Eleni A., et al.
  Julkaistu: (2011)
• Enhanced Angiogenesis and Reduced Contraction in Thrombospondin-2–null Wounds Is Associated With Increased Levels of Matrix Metalloproteinases-2 and -9, and Soluble VEGF
  Tekijä: MacLauchlan, Susan, et al.
  Julkaistu: (2009)
• Proteolysis of Cell-Surface Tissue Transglutaminase by Matrix Metalloproteinase-2 Contributes to the Adhesive Defect and Matrix Abnormalities in Thrombospondin-2-Null Fibroblasts and Mice
  Tekijä: Agah, Azin, et al.
  Julkaistu: (2005)