Enhanced Angiogenesis and Reduced Contraction in Thrombospondin-2–null Wounds Is Associated With Increased Levels of Matrix Metalloproteinases-2 and -9, and Soluble VEGF

Ítems similars

• Proteolysis of Cell-Surface Tissue Transglutaminase by Matrix Metalloproteinase-2 Contributes to the Adhesive Defect and Matrix Abnormalities in Thrombospondin-2-Null Fibroblasts and Mice
  per: Agah, Azin, et al.
  Publicat: (2005)
• Thrombospondin-2 Modulates Extracellular Matrix Remodeling during Physiological Angiogenesis
  per: Krady, Marie M., et al.
  Publicat: (2008)
• The Lack of Thrombospondin-1 (TSP1) Dictates the Course of Wound Healing in Double-TSP1/TSP2-Null Mice
  per: Agah, Azin, et al.
  Publicat: (2002)
• Matricellular Proteins as Modulators of Cell–Matrix Interactions: Adhesive Defect in Thrombospondin 2-null Fibroblasts is a Consequence of Increased Levels of Matrix Metalloproteinase-2
  per: Yang, Zhantao, et al.
  Publicat: (2000)
• Altered Extracellular Matrix Remodeling and Angiogenesis in Sponge Granulomas of Thrombospondin 2-Null Mice
  per: Kyriakides, Themis R., et al.
  Publicat: (2001)