Proteolysis of Cell-Surface Tissue Transglutaminase by Matrix Metalloproteinase-2 Contributes to the Adhesive Defect and Matrix Abnormalities in Thrombospondin-2-Null Fibroblasts and Mice

Samankaltaisia teoksia

• Matricellular Proteins as Modulators of Cell–Matrix Interactions: Adhesive Defect in Thrombospondin 2-null Fibroblasts is a Consequence of Increased Levels of Matrix Metalloproteinase-2
  Tekijä: Yang, Zhantao, et al.
  Julkaistu: (2000)
• Enhanced Angiogenesis and Reduced Contraction in Thrombospondin-2–null Wounds Is Associated With Increased Levels of Matrix Metalloproteinases-2 and -9, and Soluble VEGF
  Tekijä: MacLauchlan, Susan, et al.
  Julkaistu: (2009)
• The Lack of Thrombospondin-1 (TSP1) Dictates the Course of Wound Healing in Double-TSP1/TSP2-Null Mice
  Tekijä: Agah, Azin, et al.
  Julkaistu: (2002)
• Altered Extracellular Matrix Remodeling and Angiogenesis in Sponge Granulomas of Thrombospondin 2-Null Mice
  Tekijä: Kyriakides, Themis R., et al.
  Julkaistu: (2001)
• Up-regulation of Thrombospondin-2 in Akt1-null Mice Contributes to Compromised Tissue Repair Due to Abnormalities in Fibroblast Function
  Tekijä: Bancroft, Tara, et al.
  Julkaistu: (2015)