Blocking TGF-β–Smad2/3 innate immune signaling mitigates Alzheimer-like pathology

Alzheimer's disease is the most common dementia and is pathologically characterized by deposition of amyloid-β peptide (Aβ) into β-amyloid plaques, neuronal injury and low-level, chronic activation of brain immunity1. Transforming growth factor-βs (TGF-βs) are pleiotropic cytokines that have ke...

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Detaylı Bibliyografya
Asıl Yazarlar: Town, Terrence, Laouar, Yasmina, Pittenger, Christopher, Mori, Takashi, Szekely, Christine A, Tan, Jun, Duman, Ronald S, Flavell, Richard A
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2008
Konular:
Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC2649699/
https://ncbi.nlm.nih.gov/pubmed/18516051
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/nm1781
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