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Tumor Necrosis Factor-α Potentiates Intraneuronal Ca(2+) Signaling via Regulation of the Inositol 1,4,5-Trisphosphate Receptor
Inflammatory events have long been implicated in initiating and/or propagating the pathophysiology associated with a number of neurological diseases. In addition, defects in Ca(2+)-handling processes, which shape membrane potential, influence gene transcription, and affect neuronal spiking patterns,...
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| Päätekijät: | , , |
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| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
American Society for Biochemistry and Molecular Biology
2008
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2586262/ https://ncbi.nlm.nih.gov/pubmed/18838384 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M802209200 |
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