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Sustained NF-κB activation produces a short-term cell proliferation block in conjunction with repressing effectors of cell cycle progression controlled by E2F or FoxM1
NF-κB transcription factors induce a host of genes involved in pro-inflammatory/stress-like responses; but the collateral effects and consequences of sustained NF-κB activation on other cellular gene expression programming remain less well understood. Here enforced expression of a constitutively act...
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| Huvudupphovsmän: | , , , , , , , , |
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| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
2009
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2581928/ https://ncbi.nlm.nih.gov/pubmed/18803232 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/jcp.21596 |
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