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WNK3 and WNK4 amino-terminal domain defines their effect on the renal Na(+)-Cl(−) cotransporter

Loss of physiological regulation of the renal thiazide-sensitive Na(+)-Cl(−) cotransporter (NCC) by mutant WNK1 or WNK4 results in pseudohypoaldosteronism type II (PHAII) characterized by arterial hypertension and hyperkalemia. WNK4 normally inhibits NCC, but this effect is lost by eliminating WNK4...

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Bibliografski detalji
Glavni autori: San-Cristobal, Pedro, Ponce-Coria, José, Vázquez, Norma, Bobadilla, Norma A., Gamba, Gerardo
Format: Artigo
Jezik:Inglês
Izdano: American Physiological Society 2008
Teme:
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC2576145/
https://ncbi.nlm.nih.gov/pubmed/18701621
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.90396.2008
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