Inhibition of apoB secretion from HepG2 cells by insulin is amplified by naringenin, independent of the insulin receptor

Hepatic overproduction of apolipoprotein B (apoB)-containing lipoproteins is characteristic of the dyslipidemia associated with insulin resistance. Recently, we demonstrated that the flavonoid naringenin, like insulin, decreased apoB secretion from HepG2 cells by activation of both the phosphoinosit...

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Main Authors: Allister, Emma M., Mulvihill, Erin E., Barrett, P. Hugh R., Edwards, Jane Y., Carter, Lindsey P., Huff, Murray W.
Formáid: Artigo
Teanga:Inglês
Foilsithe: American Society for Biochemistry and Molecular Biology 2008
Ábhair:
Research Article
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC2533411/
https://ncbi.nlm.nih.gov/pubmed/18587069
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1194/jlr.M800297-JLR200
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