Inhibition of apoB secretion from HepG2 cells by insulin is amplified by naringenin, independent of the insulin receptor

Hepatic overproduction of apolipoprotein B (apoB)-containing lipoproteins is characteristic of the dyslipidemia associated with insulin resistance. Recently, we demonstrated that the flavonoid naringenin, like insulin, decreased apoB secretion from HepG2 cells by activation of both the phosphoinosit...

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Detalhes bibliográficos
Main Authors: Allister, Emma M., Mulvihill, Erin E., Barrett, P. Hugh R., Edwards, Jane Y., Carter, Lindsey P., Huff, Murray W.
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Biochemistry and Molecular Biology 2008
Assuntos:
Research Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2533411/
https://ncbi.nlm.nih.gov/pubmed/18587069
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1194/jlr.M800297-JLR200
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