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Phosphorylation of MDMX Mediated by Akt Leads to Stabilization and Induces 14-3-3 Binding

The critical tumor suppressor p53 is mutated or functionally inactivated in nearly all cancers. We have shown previously that the MDM2-MDMX complex functions as an integral unit in targeting p53 for degradation. Here we identify the small protein 14-3-3 as a binding partner of MDMX, which binds at t...

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Bibliografiske detaljer
Main Authors: Lopez-Pajares, Vanessa, Kim, Mihee M., Yuan, Zhi-Min
Format: Artigo
Sprog:Inglês
Udgivet: American Society for Biochemistry and Molecular Biology 2008
Fag:
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2376244/
https://ncbi.nlm.nih.gov/pubmed/18356162
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M710030200
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