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RelA/p65 promotes osteoclast differentiation by blocking a RANKL-induced apoptotic JNK pathway in mice

Osteoclasts (OCs) function to reabsorb bone and are responsible for the bone loss associated with inflammatory arthritis and osteoporosis. OC numbers are elevated in most disorders of accelerated bone destruction, reflecting altered rates of precursor differentiation and apoptosis. Both of these pro...

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Autori principali: Vaira, Sergio, Alhawagri, Muhammad, Anwisye, Imani, Kitaura, Hideki, Faccio, Roberta, Novack, Deborah Veis
Natura: Artigo
Lingua:Inglês
Pubblicazione: American Society for Clinical Investigation 2008
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC2373419/
https://ncbi.nlm.nih.gov/pubmed/18464930
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI33392
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