Inhibiting transthyretin conformational changes that lead to amyloid fibril formation
Insoluble protein fibrils resulting from the self-assembly of a conformational intermediate are implicated as the causative agent in several severe human amyloid diseases, including Alzheimer’s disease, familial amyloid polyneuropathy, and senile systemic amyloidosis. The latter two diseases are ass...
में बचाया:
| में प्रकाशित: | Proc Natl Acad Sci U S A |
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| मुख्य लेखकों: | , , , , , |
| स्वरूप: | Artigo |
| भाषा: | Inglês |
| प्रकाशित: |
National Academy of Sciences
1998
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| विषय: | |
| ऑनलाइन पहुंच: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC23669/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9789022/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.95.22.12956 |
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